A client with glomerulonephritis experiences hypertension as a symptom. What is the underlying mechanism that contributes to the development of hypertension in this condition?

Increased renin production

Decreased glomerular filtration rate (GFR)

Elevated blood glucose levels

Hypercalcemia

Answer and Explanation

The Correct Answer is A

A) Correct. In glomerulonephritis, renal damage can lead to increased renin production, which in turn raises blood pressure.

B) Incorrect. Glomerulonephritis may affect GFR, but decreased GFR alone is not the primary cause of hypertension in this condition.

C) Incorrect. Elevated blood glucose levels are associated with diabetes mellitus but are not the primary cause of hypertension in glomerulonephritis.

D) Incorrect. Hypercalcemia (high blood calcium levels) is not a common cause of hypertension in glomerulonephritis.

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Correct Answer is C

Explanation

A) Incorrect. Glomerulonephritis is not primarily characterized by reduced blood flow to the kidneys.

B) Incorrect. Increased GFR is not the primary mechanism involved in the pathophysiology of glomerulonephritis.

C) Correct. Inflammation and damage to the glomerular basement membrane, often triggered by immune responses, are central to the pathophysiology of glomerulonephritis.

D) Incorrect. Excessive reabsorption of sodium is not a primary factor in the development of glomerulonephritis.

A. Elevating the legs while sitting or lying down. B. Increasing sodium intake to maintain electrolyte balance. C. Limiting daily fluid intake to less than 500 ml. D. Avoiding any movement to reduce fluid accumulation.

Correct Answer is A

Explanation

A) Correct. Elevating the legs can help reduce edema by promoting venous return and decreasing fluid accumulation in the lower extremities.

B) Incorrect. Increasing sodium intake is not advisable as it can exacerbate edema and hypertension.

C) Incorrect. Severely limiting daily fluid intake is not recommended without medical guidance and can lead to dehydration.

D) Incorrect. Avoiding all movement is not necessary and may not effectively reduce fluid accumulation in edematous areas.

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A client with glomerulonephritis experiences hypertension as a symptom. What is the underlying mechanism that contributes to the development of hypertension in this condition?

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Naxlex Comprehensive Predictor Exams

A nurse is explaining the pathophysiology of glomerulonephritis to a client. What is the primary underlying process that occurs in glomeruli leading to this condition?

Correct Answer is C

Explanation

A client with glomerulonephritis is experiencing edema. What self-care measure can the nurse recommend to help alleviate edema at home?

Correct Answer is A

Explanation

A nurse is discussing the potential complications of glomerulonephritis with a client. Which complication is related to the formation of crescent-shaped structures within the glomeruli?

A nurse is caring for a client with glomerulonephritis who reports a decreased urine output. Which medical term should the nurse use to describe this symptom?

A client with glomerulonephritis experiences hypertension as a symptom. What is the underlying mechanism that contributes to the development of hypertension in this condition?

A nurse is educating a client about the diagnostic evaluation of glomerulonephritis. Which laboratory finding is commonly associated with glomerulonephritis and is characterized by the presence of red blood cells in the urine?

A client with glomerulonephritis complains of fatigue, weakness, and pallor. What is the most likely cause of these symptoms?

A nurse is educating a client about the risk factors for glomerulonephritis. Which lifestyle factor is often associated with an increased risk of chronic glomerulonephritis?

A nurse is explaining to a client the importance of a kidney biopsy in diagnosing glomerulonephritis. What is the primary purpose of a kidney biopsy in this context?

A nurse is discussing the role of the immune system in glomerulonephritis with a client. Which immune response is commonly associated with the development of immune complex-mediated glomerulonephritis?