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Aspirin may be used for DVT prophylaxis in low-risk cases, but it is less effective than anticoagulants like enoxaparin after major orthopedic surgery. Total knee replacement carries a high DVT risk, requiring stronger prophylaxis, making aspirin a less preferred choice for this scenario.

Warfarin is an anticoagulant but requires days to reach therapeutic levels, making it less ideal for immediate postoperative DVT prevention. Enoxaparin acts quickly, providing effective prophylaxis after knee replacement, making warfarin less suitable and incorrect for this context.

Alteplase, a thrombolytic, dissolves existing clots but is not used for DVT prevention. It increases bleeding risk, inappropriate for routine postoperative prophylaxis, making this choice incorrect compared to enoxaparin’s preventive mechanism.

Slow, steady bubbling in the suction control chamber is normal, indicating proper suction function, not an air leak. Checking for leaks is unnecessary unless bubbling is vigorous or in the water seal chamber, making this choice incorrect for the observed finding.

Notifying the provider is unwarranted, as slow, steady bubbling in the suction control chamber is an expected finding post-thoracotomy. It reflects normal system function, not a complication, making this choice unnecessary compared to continued monitoring.

Increasing suction pressure is inappropriate, as slow, steady bubbling suggests adequate suction. Excessive pressure risks lung damage or system malfunction, making this choice incorrect and potentially harmful for the client’s chest drainage system.

Documenting is important but not the first action. No bubbling in the suction control chamber suggests a suction failure, requiring immediate correction to maintain chest drainage, making this choice secondary to checking the system.

Adding water is incorrect, as the suction control chamber’s water level regulates suction, not its presence. No bubbling indicates a suction source issue, not low water, making this choice inappropriate for the problem.

Milking the tubing is outdated and risks dislodging clots or damaging lung tissue. The issue is suction failure, not tubing obstruction, making this choice incorrect and potentially harmful for the client.

A low-salt diet is relevant for heart failure, not COPD. It does not address lung congestion directly, as sodium restriction primarily manages fluid retention, making this choice incorrect for the client’s issue.

Delivering oxygen at 2 L/min may be used in COPD but is not the first action for congestion. Hydration directly addresses mucus, while oxygen targets hypoxemia, making this choice less relevant.

Frequent position changes aid mobility but do not directly relieve lung congestion. Hydration thins mucus, improving clearance, making this choice less effective than encouraging fluid intake for the client’s symptoms.

Notifying the provider is important but not the first action. The client’s decision to leave AMA requires immediate documentation via an AMA form to protect legally and ensure clarity, making this secondary.

Explaining risks is valuable but does not address the legal requirement of documenting AMA discharge. The AMA form ensures the client’s decision is recorded, making this choice secondary to form signing.

Requesting security is inappropriate, as competent clients can refuse care. Forcing the client to stay violates autonomy, and the AMA form is the correct step, making this choice incorrect.

Droplet precautions address respiratory infections, not low platelets. Leukemia-related bleeding risk is unrelated to droplet-transmitted pathogens, making this choice incorrect for the child’s thrombocytopenia.

Neutropenic precautions apply to low white blood cell counts, not platelets. While leukemia may cause neutropenia, the question specifies low platelets, making bleeding precautions the priority, so this choice is incorrect.

Contact precautions prevent infection spread, like MRSA, but do not address bleeding risk from low platelets. The child’s thrombocytopenia requires specific bleeding precautions, making this choice incorrect.

Aspirin’s anti-inflammatory effect reduces inflammation but is not the primary reason for MI treatment. Its antiplatelet action prevents clot formation, critical for preventing further coronary occlusion. Inflammation control is secondary, making this less relevant for aspirin’s role in MI management.

Aspirin’s antipyretic effect reduces fever, irrelevant in MI unless fever is present. Its antiplatelet effect is critical for preventing thrombus progression, directly addressing the pathophysiology of MI, making fever reduction an incorrect primary action for this context.

Aspirin’s analgesic effect relieves pain, but MI pain is managed with other medications (e.g., nitroglycerin, morphine). The antiplatelet effect is the primary reason for its use, preventing further clotting, making pain relief a secondary consideration in MI treatment.