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Infiltration. This is when the IV fluid leaks into the surrounding tissue, causing swelling, coolness, and pallor. This does not match the symptoms of redness, warmth, and tenderness.

Extravasation. This is when the IV fluid is a vesicant, meaning it can cause tissue damage or necrosis if it leaks into the surrounding tissue. This can cause pain, burning, blistering, or skin sloughing. This does not match the symptoms of redness, warmth, and tenderness.

Thrombophlebitis. This is when a blood clot forms in the vein, causing inflammation and obstruction. This can cause pain, swelling, redness, warmth, and a palpable cord along the vein. However, this is more likely to occur in larger veins such as the femoral or saphenous veins, not in the cephalic vein.

This is incorrect because a burning sensation at the site is more likely to indicate phlebitis, which is inflammation of the vein, rather than infiltration, which is leakage of fluid into the surrounding tissue.

This is incorrect because blood leaking from the site is more likely to indicate a hematoma, which is a collection of blood outside the blood vessel, rather than infiltration.

This is incorrect because difficulty moving the arm is more likely to indicate nerve damage or compartment syndrome, which are serious complications of IV therapy, rather than infiltration.

Fever and chills are not signs of extravasation, but rather of infection or an allergic reaction to the antineoplastic medication.

Nausea and vomiting are common side effects of antineoplastic medications, but they are not caused by extravasation. Extravasation is the leakage of the medication into the surrounding tissues.

Hypotension and tachycardia are not specific signs of extravasation, but rather of shock or hemorrhage. The nurse should monitor the client's vital signs and report any abnormal findings to the primary health care provider.

Dehydration is not a risk factor for thrombophlebitis, which is inflammation of a vein due to a blood clot. Dehydration can cause hemoconcentration, which increases the risk of thrombosis, but not necessarily thrombophlebitis.

Hyperglycemia is not a risk factor for thrombophlebitis, although it can increase the risk of other vascular complications, such as atherosclerosis and peripheral arterial disease. Hyperglycemia can damage the endothelial lining of the blood vessels, but this does not directly cause thrombophlebitis.

Hypertension is not a risk factor for thrombophlebitis, although it can increase the risk of other cardiovascular problems, such as stroke and heart failure. Hypertension can increase the pressure and stress on the arterial walls, but this does not affect the venous system where thrombophlebitis occurs.